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Markers of neuroplasticity including dendritic spine density and synaptic protein expression remain elevated for 10-14 days following administration, meaning the neuroplastic processes initiated by earlier doses continue developing during the interruption

Together these mechanistic effects make 2APB are very potent antidote against APAP toxicity, which is likely totally independent of gap junctions as connexin 32 knockdown mice do not show consistent protection [156, 157]

At the molecular level, specific constituents contribute to its anti-inflammatory profile

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