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G-actin sequestration Primary function, essential for motility of fibroblasts, keratinocytes, endothelial cells and cardiomyocytes

concurrently, BNIP3 recruits Parkin to mitochondria to initiate autophagic flux, whereas NIXas a ubiquitination substrate of Parkinundergoes conformational remodeling post-modification to specifically engage adaptor protein NBR1, ultimately enabling targeted autophagic degradation of compromised mitochondria (Wang et al., 2023)

It may support: Cartilage regeneration Joint inflammation reduction Pain relief during physical activity This makes it a compelling option for individuals with joint pain or conditions like osteoarthritis, improving both mobility and exercise sustainability 2